HAEM5:Erdheim-Chester disease: Difference between revisions

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{{DISPLAYTITLE:Erdheim-Chester disease}}
[[HAEM5:Table_of_Contents|Haematolymphoid Tumours (WHO Classification, 5th ed.)]]
[[HAEM5:Table_of_Contents|Haematolymphoid Tumours (WHO Classification, 5th ed.)]]


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==Primary Author(s)*==
==Primary Author(s)*==


Put your text here<span style="color:#0070C0"> (''<span class="blue-text">EXAMPLE:</span>'' Jane Smith, PhD) </span>
Mayuri Shende, MBBS, DCP, FCPS, DNB, ASCP-SH CM
 
Scott Turner, PhD<span style="color:#0070C0"> (''<span class="blue-text">EXAMPLE:</span>'' Jane Smith, PhD) </span>


__TOC__
__TOC__
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==Definition / Description of Disease==
==Definition / Description of Disease==


Put your text here <span style="color:#0070C0">(''Instructions: Brief description of approximately one paragraph - include disease context relative to other WHO classification categories, diagnostic criteria if applicable, and differential diagnosis if applicable. Other classifications can be referenced for comparison.'') </span>
Erdheim–Chester disease (ECD) is a histiocytic neoplasm characterized by accumulation of mature histiocytes, associated with inflammation and fibrosis in many different organs . <span style="color:#0070C0">(''Instructions: Brief description of approximately one paragraph - include disease context relative to other WHO classification categories, diagnostic criteria if applicable, and differential diagnosis if applicable. Other classifications can be referenced for comparison.'') </span>


==Synonyms / Terminology==
==Synonyms / Terminology==


Put your text here <span style="color:#0070C0">(''Instructions: Include currently used terms and major historical ones, adding “(historical)” after the latter.'') </span>
Erdheim–Chester disease (ECD) <span style="color:#0070C0">(''Instructions: Include currently used terms and major historical ones, adding “(historical)” after the latter.'') </span>


==Epidemiology / Prevalence==
==Epidemiology / Prevalence==


Put your text here
Rare histiocytic neoplasm with median age at diagnosis being 55 years, male predilection. Many cases might be underdiagnosed.


==Clinical Features==
==Clinical Features==
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{| class="wikitable"
{| class="wikitable"
|'''Signs and Symptoms'''
|'''Signs and Symptoms'''
|<span class="blue-text">EXAMPLE:</span> Asymptomatic (incidental finding on complete blood counts)
|Asymptomatic or presents with fever, fatigue
Site related symptoms like bone pain, exophthalmos, xanthelasma, diabetes insipidus


<span class="blue-text">EXAMPLE:</span> B-symptoms (weight loss, fever, night sweats)
CNS-mass effect or neurodegerative type symptoms


<span class="blue-text">EXAMPLE:</span> Fatigue
Cardiac tamponade


<span class="blue-text">EXAMPLE:</span> Lymphadenopathy (uncommon)
Painful abdominal mass
|-
|-
|'''Laboratory Findings'''
|'''Laboratory and imaging Findings'''
|<span class="blue-text">EXAMPLE:</span> Cytopenias
|Symmetrical osteosclerosis of the leg bones on PET with bilateral uptake of FDG
Hairy-kidney appearance in cases of bilateral perinephric involvement


<span class="blue-text">EXAMPLE:</span> Lymphocytosis (low level)
Mass-like infiltration of the right atrium on MRI
 
Sclerosis of sinuses of the face on CT
 
May be associated with chronic myelomonocytic leukaemia, Langerhans cell histiocytosis
|}
|}


==Sites of Involvement==
==Sites of Involvement==


Put your text here <span style="color:#0070C0">(''Instruction: Indicate physical sites; <span class="blue-text">EXAMPLE:</span> nodal, extranodal, bone marrow'') </span>
ECD can involve any organ, mostly excludes lymph nodes, spleen and liver. In majority of cases (80-95%) involves long bones, bilaterally and symmetric. Perinephric, periaortic involvement also frequent. Pitutary, neurologic, pulmonary, cardiac, serosal and cutaneous involvement (manifesting as xanthelasma or papule) also noted. <span style="color:#0070C0">(''Instruction: Indicate physical sites; <span class="blue-text">EXAMPLE:</span> nodal, extranodal, bone marrow'') </span>


==Morphologic Features==
==Morphologic Features==


Put your text here
Histopathology:
 
*Infiltration by foamy, lipid-laden, and/or small mononuclear histiocytes and variable proportions of Touton giant cells, small lymphocytes, plasma cells, and/or neutrophils.
*Fibrosis usually present
*Can be misdiagnosed as reactive fibroinflammatory process if there is extensive fibrosis or it may also show partial morphological and immunophenotypic overlap with Rosai–Dorfman disease or with reticulohistiocytosis or Langerhans cell histiocytosis.


==Immunophenotype==
==Immunophenotype==
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!Finding!!Marker
!Finding!!Marker
|-
|-
|Positive (universal)||<span class="blue-text">EXAMPLE:</span> CD1
|Positive (universal)||CD163, CD68, CD14, and CD4
Positive for factor XIIIa, fascin, and (less commonly) S100
 
Diffuse strong cytoplasmic staining with VE1 monoclonal antibody suggestive of BRAF p.V600E mutation, but should be confirmed with molecular analysis
 
Expression of phosphorylated ERK also noted frequently
|-
|-
|Positive (subset)||<span class="blue-text">EXAMPLE:</span> CD2
|Positive (subset)||<span class="blue-text">EXAMPLE:</span> CD2
|-
|-
|Negative (universal)||<span class="blue-text">EXAMPLE:</span> CD3
|Negative (universal)||CD1a and CD207
|-
|-
|Negative (subset)||<span class="blue-text">EXAMPLE:</span> CD4
|Negative (subset)||<span class="blue-text">EXAMPLE:</span> CD4
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!Gene; Genetic Alteration!!Pathway!!Pathophysiologic Outcome
!Gene; Genetic Alteration!!Pathway!!Pathophysiologic Outcome
|-
|-
|<span class="blue-text">EXAMPLE:</span> BRAF and MAP2K1; Activating mutations
|Gain-of-function somatic alteration
|<span class="blue-text">EXAMPLE:</span> MAPK signaling
|MAPK cell signalling pathway
|<span class="blue-text">EXAMPLE:</span> Increased cell growth and proliferation
|<span class="blue-text">EXAMPLE:</span> Increased cell growth and proliferation
|-
|-
|<span class="blue-text">EXAMPLE:</span> CDKN2A; Inactivating mutations
|''BRAF'' p.V600E mutations or ''BRAF'', ''ARAF'', ''NRAS'', ''KRAS'', or ''MAP2K1''
|<span class="blue-text">EXAMPLE:</span> Cell cycle regulation
|<span class="blue-text">EXAMPLE:</span> Cell cycle regulation
|<span class="blue-text">EXAMPLE:</span> Unregulated cell division
|<span class="blue-text">EXAMPLE:</span> Unregulated cell division
|-
|-
|<span class="blue-text">EXAMPLE:</span>  KMT2C and ARID1A; Inactivating mutations
|
|<span class="blue-text">EXAMPLE:</span>  Histone modification, chromatin remodeling
|''PIK3CA''
|<span class="blue-text">EXAMPLE:</span>  Abnormal gene expression program
|<span class="blue-text">EXAMPLE:</span>  Abnormal gene expression program
|}
|}
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'''EXAMPLE Book'''
'''EXAMPLE Book'''


#Arber DA, et al., (2017). Acute myeloid leukaemia with recurrent genetic abnormalities, in World Health Organization Classification of Tumours of Haematopoietic and Lymphoid Tissues, Revised 4th edition. Swerdlow SH, Campo E, Harris NL, Jaffe ES, Pileri SA, Stein H, Thiele J, Arber DA, Hasserjian RP, Le Beau MM, Orazi A, and Siebert R, Editors. IARC Press: Lyon, France, p129-171.
#John  Chan, et al., Erdheim–Chester disease in WHO Classification of Tumours Editorial Board. Haematolymphoid tumours. Lyon (France): International Agency for Research on Cancer; 2024. . (WHO classification of tumours series, 5th ed.; vol. 11). <nowiki>https://publications.iarc.who.int/637</nowiki>.


==Notes==
==Notes==
<nowiki>*</nowiki>Primary authors will typically be those that initially create and complete the content of a page.  If a subsequent user modifies the content and feels the effort put forth is of high enough significance to warrant listing in the authorship section, please contact the CCGA coordinators (contact information provided on the homepage).  Additional global feedback or concerns are also welcome.
<nowiki>*</nowiki>Primary authors will typically be those that initially create and complete the content of a page.  If a subsequent user modifies the content and feels the effort put forth is of high enough significance to warrant listing in the authorship section, please contact the CCGA coordinators (contact information provided on the homepage).  Additional global feedback or concerns are also welcome.
<nowiki>*</nowiki>''Citation of this Page'': “Erdheim-Chester disease”. Compendium of Cancer Genome Aberrations (CCGA), Cancer Genomics Consortium (CGC), updated {{REVISIONMONTH}}/{{REVISIONDAY}}/{{REVISIONYEAR}}, <nowiki>https://ccga.io/index.php/HAEM5:Erdheim-Chester_disease</nowiki>.
<nowiki>*</nowiki>''Citation of this Page'': “Erdheim-Chester disease”. Compendium of Cancer Genome Aberrations (CCGA), Cancer Genomics Consortium (CGC), updated {{REVISIONMONTH}}/{{REVISIONDAY}}/{{REVISIONYEAR}}, <nowiki>https://ccga.io/index.php/HAEM5:Erdheim-Chester_disease</nowiki>.
[[Category:HAEM5]][[Category:DISEASE]][[Category:Diseases E]]
[[Category:HAEM5]]
[[Category:DISEASE]]
[[Category:Diseases E]]