HAEM5:T-prolymphocytic leukaemia: Difference between revisions

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|inv(14)(q11.2q32.1)
|inv(14)(q11.2q32.1)
t(14;14)(q11.2;q32.1)
t(14;14)(q11.2;q32.1)
|TCL1A/TRD|| ||inv(14) ~60%
|TCL1A/B ,TRD|| ||inv(14) ~60%
t(14;14) ~25%
t(14;14) ~25%
|Yes
|Yes
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|-
|-
|t(X;14)(q28;q11.2)
|t(X;14)(q28;q11.2)
|MTCP1/TRD
|MTCP1, TRD
|
|
|Low (5%)
|Low (5%)
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|}
==Individual Region Genomic Gain / Loss / LOH==
==Individual Region Genomic Gain / Loss / LOH==
Approximately, 70-80% of T-PLL karyotypes are complex, typically containing 3-5 or more structural aberrations. Common cytogenetic abnormalities include those of chromosome 8, such as idic(8)(p11.2), t(8;8)(p11.2;q12), and trisomy 8q. Other frequent changes are deletions in 12p13 and 22q, gains in 8q24 (MYC), and abnormalities in chromosomes 5p, 6, and 17.
{| class="wikitable sortable"
{| class="wikitable sortable"
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|5
|5
|Gain/Loss
|5p
|chr5
|Yes
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|Minor diagnostic criteria
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|6
|6
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|Gain/Loss
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|6p
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|chr6
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|No
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|11
|11
|Loss
|Loss
|11q23.3
|11q
|ch11q21-q23
|ch11q21-q23.3
|Yes
|Yes
|Yes
|Yes
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|12
|12
|Loss
|Loss
|12p
|12p13
|12p13
|chr12
|Yes
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|Minor diagnostic criteria
|-
|-
|13
|13
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|Loss
|13q
|13q14.3
|13q14.3
|Yes
|
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|Minor diagnostic criteria
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|-
|14
|14
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|17
|17
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|17p, 17q
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|Loss
|Loss
|Monosomy 22
|Monosomy 22
22q
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|Minor diagnostic criteria
|}
|}
==Characteristic Chromosomal Patterns==
==Characteristic Chromosomal Patterns==
Put your text here <span style="color:#0070C0">(''EXAMPLE PATTERNS: hyperdiploid; gain of odd number chromosomes including typically chromosome 1, 3, 5, 7, 11, and 17; co-deletion of 1p and 19q; complex karyotypes without characteristic genetic findings; chromothripsis. Do not delete table.'')</span>
Approximately, 70-80% of T-PLL karyotypes are complex, typically containing 3-5 or more structural aberrations. Common cytogenetic abnormalities include those of chromosome 8, such as idic(8)(p11.2), t(8;8)(p11.2;q12), and trisomy 8q. Other frequent changes are deletions in 12p13 and 22q, gains in 8q24 (MYC), and abnormalities in chromosomes 5p, 6, and 17.
{| class="wikitable sortable"
{| class="wikitable sortable"
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!Notes
!Notes
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|-
|<span class="blue-text">EXAMPLE:</span> ''TP53''; Variable LOF mutations
|ATM
<span class="blue-text">EXAMPLE:</span>
|TSG
 
|<span class="blue-text">EXAMPLE:</span> 20% (COSMIC)
''EGFR''; Exon 20 mutations


<span class="blue-text">EXAMPLE:</span> ''BRAF''; Activating mutations
|<span class="blue-text">EXAMPLE:</span> TSG
|<span class="blue-text">EXAMPLE:</span> 20% (COSMIC)
<span class="blue-text">EXAMPLE:</span> 30% (add Reference)
<span class="blue-text">EXAMPLE:</span> 30% (add Reference)
|<span class="blue-text">EXAMPLE:</span> ''IDH1'' R123H
|<span class="blue-text">EXAMPLE:</span> ''EGFR'' amplification
|<span class="blue-text">EXAMPLE:</span> Yes
|<span class="blue-text">EXAMPLE:</span> No
|<span class="blue-text">EXAMPLE:</span> No
|<span class="blue-text">EXAMPLE:</span> Excludes hairy cell leukemia (HCL) (add reference).
|-
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|ATM
|TSG
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|ATM mutations
|ATM mutations
|None specified
|None specified
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|The mutations within this pathway typically occur in a mutually exclusive manner (Dr jaffe book)
|The mutations within this pathway typically occur in a mutually exclusive manner (Dr jaffe book)
|-
|-
|TP53
|''TP53''
|TSG
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|''BCOR''
|TSG
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!Gene; Genetic Alteration!!Pathway!!Pathophysiologic Outcome
!Gene; Genetic Alteration!!Pathway!!Pathophysiologic Outcome
|-
|-
|''TCL1A rearrangement''
|''TCL1A/B rearrangement''
|''AKT'' signaling and TCR signal amplification pathways
|''AKT'' signaling and TCR signal amplification pathways
|Increased cell survival and proliferation
|Increased cell survival and proliferation